Datasets

subject: Biomedical Engineering

Total is 44 Results
Changes in Lipid Profile and SOX-2 Expression in RM-1 Cells after Co-Culture with Preimplantation Embryos or with Deproteinated Blastocyst Extracts

10.4231/RVM1-G302

Bennett D. Elzey , Bruce R. Cooper , Christina R. Ferreira ORCID logo , Gregory M. Cresswell , Judy E. Hallett , Nicolas Gutierrez ORCID logo , Wen-Hung Wang

03/08/2022

The embryonic environment can modify cancer cell metabolism, and it is reported to induce the loss of tumorigenic properties and even affect the differentiation of cancer cells into normal tissues. The cellular mechanisms related to this...

Biomedical Engineering cancer cell metabolism embryonic environment lipid profiling RM-1 cells SOX-2 gene expression

Error-State Kalman Filter for Online Evaluation of Ankle Angle

10.4231/X00Q-YW81

Ahmed Khaled Soliman , Andres Torres , Guilherme A. Ribeiro , Mo Rastgaar ORCID logo

05/04/2022

Raw Data for an Error-State Kalman Filter (ESKF) for Online Evaluation of Ankle Ankle (in 2-DOF), using a 2-IMU setup.

Acceleration Biomedical Engineering Engineering Technology Matlab software Mechanical Engineering Robotics

A Multi-state Model of the CaMKII Holoenzyme using MCell 3.3

10.4231/MV0Z-8Z57

Matthew C Pharris , Tamara L Kinzer-Ursem ORCID logo

07/29/2019

This model uses a specialized rule-based syntax in MCell 3.3 to model the twelve-subunit CaMKII holoenzyme without inducing combinatorial explosion. The model allows us to explore the regulation of CaMKII activation and autophosphorylation.

Biomedical Engineering Calcium Calmodulin Computational Modeling Kinase Neuroscience Protein Signaling Rule-Based Modeling Synaptic Plasticity

MicroCT based FE model of bone core with tissue heterogeneity and anisotropy

10.4231/R7CC0XX4

Joseph Wallace , Matthew R Allen , Max A Hammond , Thomas Siegmund ORCID logo

06/26/2018

This publication contains a finite element model for the analysis of bone core under consideration of bone tissue heterogeneity and tissue anisotropy.

Biomedical Engineering Bone Finite Element Analysis Mechanical Engineering

Competitive Tuning of Ca2+/Calmodulin-Activated Proteins Provides a Compensatory Mechanism for AMPA Receptor Phosphorylation in Synaptic Plasticity

10.4231/R7VX0DS0

Matthew C Pharris , Tamara L. Kinzer-Ursem ORCID logo

07/30/2018

Code for the basic 4-state competitive binding model that builds on previous work by incorporating an additional competitor for calmodulin along with a number of downstream proteins. Also include is sample code for global sensitivity analysis...

Biomedical Engineering Calmodulin Neuroscience

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